By John R Hodges
There were huge, immense advances during the last decade in our realizing of cognitive functionality, relatively elements of reminiscence, language and a spotlight, yet those haven't been made available to clinicians. This ebook goals to include those advances in thought into scientific perform and to supply a realistic method of cognitive valuation on the bedside, in line with equipment constructed on the Cambridge hospital over the last 15 years. Designed basically for neurologists, psychiatrists and geriatricians in education who require a realistic consultant to assessing larger psychological functionality, the publication may also be of curiosity to medical psychologists.
In this long-awaited moment variation, John Hodges has considerably re-organized and elevated at the unique textual content. The publication encompasses a new bankruptcy dedicated to the Revised model of the Addenbrooke's Cognitive exam (ACE-R), with an outline of its makes use of and barriers besides normative information. Given the significance of the early detection of dementia a bankruptcy is devoted to this subject which pulls on advances during the last decade. a number of new illustrative case histories have additionally been extra and all the case descriptions were oriented round the use of the ACE-R in medical practice.
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Extra resources for Cognitive Assessment for Clinicians
However, many neurologists and psychiatrists work without adequate neuropsychological provision. By becoming more conversant with bedside cognitive testing, clinicians should be able to use the services of their neuropsychologists more effectively. It is not necessary, for instance, to refer every patient with suspected dementia; many patients can be satisfactorily diagnosed by clinicians if the basic principles outlined in the book are followed. There are, however, patients in whom a thorough neuropsychological evaluation is mandatory, as is illustrated by several of the cases in Chapter 5.
The apparent tail-off in very late life is probably due to under-reporting. The patient, who is generally in good health, suddenly becomes profoundly amnesic. Short-term (working) memory is preserved, but he or she is unable to retain any new information for more than a few seconds. The profound anterograde amnesia is accompanied by a variable retrograde memory deficit spreading back weeks, months, or even years. ’) but there is no impairment of conscious level or attention, and no language or visuo-spatial deficits.
1. Hence abnormalities of these distributed functions do not, with a few notable exceptions (such as amnesia following thalamic strokes), arise from small discrete lesions, but typically result from fairly extensive and often bilateral damage or more generalized insults of the type encountered in general medical practice. Localized functions can, in turn, be divided into those associated with the dominant, usually left, hemisphere, and those associated with the non-dominant hemisphere. In this chapter, I shall describe three broad domains of cognition that have a distributed neural basis: arousal/attention, memory and executive function.
Cognitive Assessment for Clinicians by John R Hodges