By Edward R. Laws Jr., Andrew H. Kaye
Meet the expanding desire for powerful mind tumor administration with the hugely expected revision of mind Tumors by means of Drs. Andrew H. Kaye and Edward R. legislation. over the last decade, huge, immense advances were made in either the analysis and the surgical and radiotherapeutic administration of mind tumors. This re-creation courses you thru the newest advancements within the box, together with sizzling subject matters like malignant gliomas, useful mind mapping, neurogenetics and the molecular biology of mind tumors, and biologic and gene treatment. You'll even have quick access to the whole contents on-line, with hyperlinks to PubMed and a downloadable photo library, at www.expertconsult.com.
-Benefit from the information and event of Drs. Andrew H. Kaye and Edward R. legislation, globally well-known specialists within the box of neurosurgery, in addition to many different global authorities.
Access the entire searchable contents on-line at www.expertconsult.com, with hyperlinks to PubMed and a downloadable photo library.
-Stay brand new with the newest advancements within the box, together with administration of
malignant gliomas; useful mind mapping; neurogenetics and the molecular biology of mind tumors; biologic and gene remedies; and masses more.
Apply the expert's top practices with their key issues .
The specialist information of Drs. Kaye and legislation permits you to successfully take care of the expanding occurrence of mind tumors, from analysis to surgical and radiotherapeutic administration.
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Additional resources for Brain Tumors: An Encyclopedic Approach (3rd Edition)
The characteristic phenotype and ventricular location of these tumors suggests that the neurocytoma might arise by the transformation of neuronally biased, transit-amplifying progenitor cells located in the adult sub-ventricular zone (Sim et al 2006). 31 Atypical central neurocytoma. (Left) In atypical central neurocytomas, β-III tubulin immunoreactivity is not as diffuse and is present in small cells with delicate processes. ) (Right) In the same atypical central neurocytoma, there is a marked increased in GFAP immunoreactive cells with similar cytoarchitecture as the cells containing, β-III tubulin.
32 Pediatric pilocytic astrocytoma. (A) Pilocytic astrocytoma arising in the posterior fossa showing the typical biphasic pattern of tumor cells (H&E). (B) OLIG2 immunoreactivity in the same tumor is conspicuous and particularly high in the less stellate cells. 34 Pediatric pilocytic astrocytoma. DCX immunoreactivity is most conspicuous in the cell bodies and delicate processes of the more stellate cells. 33 Pediatric pilocytic astrocytoma. (A) The elongated tumor cells may be a conspicuous component of the tumors that may form variably sized bundles of processes (H&E).
TUJ1 avidin–biotin immunoperoxidase. (B) Neurofilament epitopes (H/M NFP) are readily detectable in the same polygonal cell populations. ) example, ectopic EGFR stimulates the proliferation and inhibits the differentiation of oligodendrocyte progenitors and consequently, oligodendrocyte progenitor-like cells generate hyperplasia in the white matter (Ivkovic et al 2008). In addition, PDGFRα-positive adult neural stem cells in the SVZ generate oligodendrocytes in vivo. PDGF infused in the ventricle of adult mice induces massive SVZ stem cell proliferation and large glioma-like hyperplasias with expression of markers for astrocytes but not oligodendrocytes (Jackson et al 2006).
Brain Tumors: An Encyclopedic Approach (3rd Edition) by Edward R. Laws Jr., Andrew H. Kaye