By Michel Burnier, Robert W. Schrier (auth.), Alberto Amerio, Pasquale Coratelli, Vito M. Campese, Shaul G. Massry (eds.)
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Extra resources for Acute Renal Failure: Clinical and Experimental
Adv. Fritz, D. N. release by leukocytic pyrogen Engl. Secrist, J. Med. 308: 553 (1983). InhIbitors of prostagiandin synthesis or cathepsin B prevent muscle wasting due to sepsis in the rat, 34. 52 G. L. Bllbrey, G. R. J. Clin. Faloona, Invest. M. G. 73: White, 1483 (1984). J. P. Knochel: Hyperglucagonemia of renal fallure. 53: 35. 36. C. E. D. 841 J. Clin. Invest. (1974). Mondon, C. B. Dolkas, G. M. Reaven, P. Alto, M. Field, The site of InsulIn resistance In acute uremla, J. Diabetes Assoc. Am.
Pathophysiology of altered glomerular funetion in aminoglyeoside-treated rats. KIDNEY INT. 19: 288-296, 1981. Williams, R. , Thomas, C. , Mavar, L. G. and Evan A. : Hemodynamie and single nephron funetion during the maintenanee phase of isehemie aeute renal failure in the dog. KIDNEY INT. 19: 503-515, 1981. Venkatehalam, M. A. and Rennke, H. : The struetural and moleeular basis of glomerular filtration. CIRC. RES. 43: 337-347, 1978. Ren kin , E. M. and Gilmore, J. : Glomerular filtration. In: HANDBOOK OF PHYSIOLOGY, Seetion 8, Renal Physiology; edited by: Orloff, J.
These data clearly indicate that glucocorticoids playa major role in the enhanced muscle protein degradation of acute uremia. Moreover the increased urea-N appearance suggests that amino acids, released from skeletal muscle, are used for gluconeogenesis in the liver with a consecutive rise of hepatic ureogenesis. 46 Finally, the high levels of ionized calcium in the BN/ADX animals led us to assume that glucocorticoids might be involved in the PTH-resistance of the bone in uremia 57 Unfortunately, .
Acute Renal Failure: Clinical and Experimental by Michel Burnier, Robert W. Schrier (auth.), Alberto Amerio, Pasquale Coratelli, Vito M. Campese, Shaul G. Massry (eds.)